![]() ![]() Peak epinephrine levels to insulin-induced hypoglycemia were significantly correlated with peak cortisol levels. ![]() With the cutoff level of peak epinephrine for defining severe impairment set at 400 pg/ml, more patients with secondary adrenal insufficiency showed severe impairment of the epinephrine response than did those without it. ![]() Results: The study patients showed diminished response of plasma epinephrine to insulin-induced hypoglycemia. An insulin tolerance test was performed and serum catecholamines as well as plasma GH and serum cortisol were measured. Methods: The study population comprised 23 patients with pituitary adenoma (non-functioning adenoma or prolactinoma). The aim of this study was to determine whether the response of catecholamine secretion to hypoglycemia is disrupted in patients with pituitary adenoma. ![]() Insulin-induced hypoglycemia is used for evaluating GH–IGF-I and ACTH–adrenal axes in patients with pituitary disorders. Objective: Hypoglycemia induces rapid secretion of counterregulatory hormones such as catecholamine, glucagon, cortisol, and GH. ![]()
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